Faculty & Staff

In chronic neurodegenerations like glaucoma and Alzheimer’s disease, neurons undergo a protracted decline before they are irreversibly lost. As the disease progresses, they gradually lose the ability to receive, process, and send neuronal signals. This means there is a therapeutic window where restoring function to these “sick” neurons can slow, stop or even reverse some of the clinical deficits in these devastating conditions. My lab’s research focuses on identifying these early changes in neuron structure and function, determining the mechanisms underlying those changes, and developing therapies targeting those processes.